RT Journal
A1 Barsotti MM, Targovnik JH, Verso TA
T1 THyrotoxicosis, hypercalcemia, and secondary hyperparathyroidism
JF Archives of Internal Medicine
JO Archives of Internal Medicine
YR 1979
FD June 1
VO 139
IS 6
SP 661
OP 663
DO 10.1001/archinte.1979.03630430039013
UL http://dx.doi.org/10.1001/archinte.1979.03630430039013
AB Two patients with hypercalcemia and hyperthyroidism had elevated levels of parathyroid hormone (PTH). When the patients were made euthyroid with appropriate medical therapy, both the levels of PTH and calcium returned to normal. Since thyroid hormone can increase tissue responsiveness to catecholamines, and since catecholamines can stimulate PTH secretion, we postulate that the elevated levels of PTH were secondary to thyrotoxicosis. In patients with coexisting hyperthyroidism and hyperparathyroidism, primary hyperparathyroidism should only be diagnosed when the patient is eumetabolic.(Arch Intern Med 139:661-663, 1979)