Special impetus to the study of this particular physiological feature has been given by the work of Cushing,1 who showed experimentally and clinically that in Cheyne-Stokes respiration associated with increased intracranial tension the blood-pressure was low during the period of apnea and high during the period of hyperpnea. J. A. E. Eyster2 demonstrated the clinical value of this fact in the differentiation of Cheyne-Stokes respiration associated with increased intracranial tension from other types. The great interest and importance of this observation justifies a report based on fifteen cases of Cheyne-Stokes respiration arising from various causes, including cerebral hemorrhage, cerebral and general arteriosclerosis, myocarditis, decompensated organic heart lesions, pneumonia, infarct of the lungs and nephritis. The tracings were obtained by the Erlanger sphygmomanometer with a pneumograph attachment.
From this table it can be seen that the findings corroborate Eyster's observation with one exception, Case 5, one of pneumonia with