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The Lung in Congestive Heart Failure

Arch Intern Med. 1961;107(3):450-459. doi:10.1001/archinte.1961.03620030138018.
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Introduction  For many years there has been a great interest in the mechanism of dyspnea in patients with heart disease. Our present concepts of the nature of dyspnea are largely attributable to the careful studies of Francis Peabody.1-3 He demonstrated that patients with severe heart failure had an increase in the minute volume of respiration at rest. The rate of breathing was increased, and the tidal volume was small, resulting in an increased ventilation of dead space. Patients with heart failure exhibited a greater increase in ventilation for a given degree of exercise than did normal persons. Peabody emphasized that this increase in ventilation at rest or on exercise did not in itself explain the dyspnea of heart failure. Patients with heart disease complained of discomfort at a level of ventilation which caused no unusual sensation in normal persons. This tendency to dyspnea was closely related to the magnitude


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The American Medical Association is accredited by the Accreditation Council for Continuing Medical Education to provide continuing medical education for physicians. The AMA designates this journal-based CME activity for a maximum of 1 AMA PRA Category 1 CreditTM per course. Physicians should claim only the credit commensurate with the extent of their participation in the activity. Physicians who complete the CME course and score at least 80% correct on the quiz are eligible for AMA PRA Category 1 CreditTM.
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