Necrosis aguda del pancreas (pancreatitis edematosa y hemorragica).

Arch Intern Med (Chic). 1940;65(2):462-463. doi:10.1001/archinte.1940.00190080244014.
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The author has written an excellent monograph. The volume is 242 pages in length and has a fine bibliography.

The following conclusions are drawn: 

  1. Sixty per cent of human beings have a common duct of Wirsung, and the control of the sphincter of Oddi depends on the biliary duodenal reflux.

  2. The physiopathologic transformation produced by diseases of the biliary system and duodenum is caused by the failure of the normal defense mechanisms. In 60 per cent of the cases of acute pancreatitis abnormality exists in the biliary tract and duodenum.

  3. One is forced to admit the multiplicity of pathogenesis, since in many cases the disease takes a lymphovascular route, while in many cases there exists a concomitant hepatobiliary disease.

  4. One admits the existence of edematous pancreatitis of the following origins: idiopathic, lymphovascular, anaphylactic and inflammatory. The so-called edematous and hemorrhagic pancreatitis must be considered as part of the process


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