The general impression prevails that considerable variation in heart size is a not infrequent occurrence in pathologic conditions, and that such variations are indicated by variation in position of the apex beat due to left ventricular involvement. This, however, is not borne out by the few definite statements which could be found in the literature. Mackenzie states that during the progress of compensation no appreciable difference in heart size could be detected. Hoffman1 states that in chronic heart disease compensation may or may not be accompanied by change in size and that size in itself is no certain indication of functional capacity, that being determined by muscle tonus. Martin2 cites a case in which teleoroentgenograms during the convalescence from pneumonia showed a diminution in size of the heart. Vaquez3 illustrates a case of myocarditis showing progressive retraction of all borders during intravenous injections of strophanthin. However, the extent of such
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